Another BAD ACTOR ; a MET derivative!

If you followed our fight for the cure, we are increasingly suggesting that, nature has given us
its laws that we need to harvest and follow to win the cure, yes you need to follow the cellular pathways and select a point of intervention to stop or amplify in order to kill bad cells.  We also have to know the nature of the environment of pathways which contain facilitators (enzymes),substrates(Homeobox) and Breakers (Rb1), repair mechanisms (BRCA), CBF(leukemias) or point of traffic signal orienting the reactions(Homeobox/CBF), and amplifiers (c-MYC, growth factor).
Bad cancers are those that:
1-Recruit genes that leads to malformations (DDR2) (FAK) (VEGF)
2-Recruit genes that restore embryonic potentials (c-MET) (STEM CELLS)
3-Recruit genes Regulating reactions or Homeobox (CBF)
4-Recruit the NOTCH, AND THE Wnt
5-KNOCKS OUT Breakers, repair and regulators, induce transcription factors or affect the miRNA or the polymerases.
6-Recruit and command Ubiquitilation machinery or the Helicases.
7-Recruit the Bcl (s)
8-recruit omnipresent co-factors ("wild gene") such as Gerb2. GAB1

It is quite apparent that  genes that are associated with a growth factor will be imitating a chronic stimulation (exposure), or a driver situation.  The c-MET is a gene that encodes a growth factor, and not just any growth factor,

"c-Met (MET or MNNG HOS Transforming gene) is a proto-oncogene that encodes a protein known as hepatocyte growth factor receptor (HGFR).^[1][2] The hepatocyte growth factor receptor protein possesses tyrosine-kinase activity.^[3] The primary single chain precursor protein is post-translationally cleaved to produce the alpha and beta subunits, which are disulfide linked to form the mature receptor."

This GROWTH FACTOR will affect all major "wild gene" and amplify them like the "CAPS LOCK" button of your computers!

The like of FGF2, HGFR is another dangerous trigger to protecting affected cells from dying!  It is found in all dangerous cancers from Melanoma to hepatoma.  Aside from Anti-MET, only IL-2, and Interferon can reach these.  Although anti-P85 could play a role!  It involves the NOTCH and the Wnt as well as the CRKL (reelin) through GAB1 the binder to Grb2!  It involves the HIF1 and VHL, and everything else dangerous !