Tuesday, March 11, 2014

Questions in Pancreatitis

1.Complications from pancreatitis results when a Cytokine storm complicate the clinical tableau,
which Cytokine?
can we stop the buckling by Early input of Hydrocortisone or Interferon?
2.Does predisposition to Pancreatitis a G protein issue (when exposed to Alcohol or Tobacco?)
3.What is the exact role of PRSS1, CFTR, and SPINK genes, thier contribution to Pancreatitis
4.Most Pancreatitis lead to fibrosis, are genes guiding fibrosis the best monitor of inflammatory resolution,
what are these genes, cytokines, Metalloproteases, Integrins?
5.The dance between PRSS1 and SPINK1 shows the importance of Stoppers or breaker
any activators (PRSS1) needs stoppers/breakers (SPINK1) or regulators in human physiology
(remember the story of Factor V in Coagulation unstoppable cascade!)
6.Is IG4 only valuable in Autoimmune Pancreatitis
7.can you "block" pancreatic enzymes while feeding the patient in acute setting?
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