It is conceptually correct that main events such as Heart attack or strokes be preceded by an outburst
or an overall increase of either an hormone or a burst of Cytokine (s). This is a critical event because this information may help with prevention. Best place to look at this time is into vascular or platelet receptors. It is also true that lipid receptors must be included in the model to include activity at the Thrombus. Another interesting component to this fact is that there are other events that follow the primary ischemic events that can be fatal. Ie, the high risk of a stroke after another stroke within the following period past a primary stroke. How to prevent the secondary event now that we have had one appears to be critical.... The CRBCM is on the task...reviewing some Cytokines/Hormone receptors and their related genes!
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