but are PKP2 also depressed or overexpressed
does the processes of metastatic diseases different than base line natural cellular displacement in terms of Vimentin, plakoglobin, and desmoplakin expression. Or are these proteins behaving the same way no matter the cellular process.
*Can these parameters translate well the activity of Taxotere.
*Can level of methylated Vimentin coincide with Taxotere resistance or susceptibility, or level of side effects in the patient being treated.
*Can these genes reliably predict Adriamycin toxicity!
*can tightening Microtubule -Vimentin interaction by Antibody slow down cancer metastatic potentials?
*Is Tuberin/Hamartin more easily evaluated in the peripheral sera
Human Papillomavirus 16 E6 Oncoprotein Interferences with Insulin Signaling Pathway by Binding to TuberinZheming Lu ET AL!
NOW DOES LEVEL OF TUBERIN REFLECT LEVEL OF ACTIVITY OF E6, TRANSFORMATION IN CERVICAL CANCER, OR RESPONSE TO PLATIN BASED CHEMOTHERAPY, OR SIDE EFFECT LINKED TO INVOLVEMENT OF INSULIN, CNS DISEASES OR SIDE EFFECTS