Hyperlipidemia "under the microscope", REDUCING RISK OF CORONARY AND BLOOD VESSEL DISEASES

-does Xanthelasma, Corneal arcus, provide clues in triglyceride deposition in the artery
-does minimal pancreatitis and similar induced inflammatory disease induced by hyperlipidemia contributes to Coronary disease
-what type of inflammatory process are produced by free fatty acids and related lipid molecules
-?associated Pulmonary hypertension
-Dysbetalipoproteinemia provides more clues to pathophysiology?
-Cytokines involved in Neurologic syndrome of Chylomicronemia or is it direct disruption of lipogenesis/lysis
-What is the role of IL-6 in all this mess, can controlling IL-1 & 6 help (ie preserve elasticity of blood vessel)  could vascular cellular preservation slow down Arteriosclerotic phenomena (preservation of elastin and related compounds)
-what is the role of cytokines in vascular transformations (AS CYTOKINES INDUCED BY FFA)
-should closer elective follow-up of the pancreas serve as a biomarker of hyperlipidemia
-should early xanthoma be allowed to alert of Coronary event due to hyperlipidemia, FFA levels?
-what is the role of TSH, and the thyroid?
-what in the urine analysis predict a coronary events (as it relates to lipid)
-What Vitamins have to do with this?  Vitamin D role?
-Eruptive Vs tuberous XANTHOMAS (FAT VS CHOLESTEROL) WHY AND WHEN?
-how to consume debris of lipolysis without causing a "Cytokine storm" or an autoimmune disease
is it through IL-6
-how does specific  medicines works to minimize cytokine storm?
-How does Ileal bypass affect the cytokines, vascular transformation, should it be routine at a certain age to reduce Coronary risk
-can sonogram of the pancreas, specific artery gives a clue on coronary disease due to lipid disruption